Page 529 - IJB-9-5

International Journal of Bioprinting 3D printability and biochemical analysis of orange peel waste

47. Qin CX, Anthonisz J, Leo CH, et al., 2020, NO• resistance, 51. Leo CH, Hart JL, Woodman OL, 2011, 3’,4’-dihydroxyflavonol
induced in the myocardium by diabetes is circumvented restores endothelium dependent relaxation in small
by the NO redox sibling, nitroxyl. Antioxid Redox Signal, mesenteric artery from rats with type 1 and type 2 diabetes.
32:60–77. Eur J Pharmacol, 659:193–198.
48. Ng HH, Leo CH, Parry LJ, 2016, Serelaxin (recombinant 52. Leo CH, Jelinic M, Ng HH, et al., 2019, Recent developments
human relaxin-2) prevents high glucose-induced endothelial in relaxin mimetics as therapeutics for cardiovascular
dysfunction by ameliorating prostacyclin production in the diseases. Curr Opin Pharmacol, 45:42–48.
mouse aorta. Pharmacol Res, 107:220–228.
53. Leo CH, Ng HH, Marshall SA, et al., 2020, Relaxin reduces
49. Ng HH, Leo CH, Prakoso D, et al., 2017, Serelaxin endothelium-derived vasoconstriction in hypertension:
treatment reverses vascular dysfunction and left ventricular Revealing new therapeutic insights. Br J Pharmacol,
hypertrophy in a mouse model of type 1 diabetes. Sci Rep, 177:217–233.
7:39604.
54. Langston-Cox A, Leo CH, Tare M, et al., 2020, Sulforaphane
https://doi.org/10.1038/srep39604 improves vascular reactivity in mouse and human arteries
50. Leo CH, Fernando D, Tran L, et al., Serelaxin treatment after “preeclamptic-like” injury. Placenta, 101:242–250.
reduces oxidative stress and increases aldehyde 55. Marshall SA, Leo CH, Girling JE, et al., 2017, Relaxin
dehydrogenase-2 to attenuate nitrate tolerance. Front treatment reduces angiotensin II-induced vasoconstriction
Pharmacol, 8:141. in pregnancy and protects against endothelial dysfunction.
https://doi.org/10.3389/fphar.2017.00141 Biol Reprod, 96:895–906.

Volume 9 Issue 5 (2023) 521 https://doi.org/10.18063/ijb.776