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Journal of Clinical and Translational Research 2024; 10(6): 348-356
Journal of Clinical and Translational Research
Journal homepage: http://www.jctres.com/en/home
ORIGINAL ARTICLE
Effect of high-intensity interval and endurance training with MitoQ on
mitochondrial dynamics in rat muscle
Soheil Aminizadeh 1 , Hamid Najafipour 2 , Yaser Masoumi-Ardakani 3 , Beydolah Shahouzehi * ,
4
Mohammad Pourranjbar 5
1 Physiology Research Center, Institute of Neuropharmacology, Department of Physiology and Pharmacology, Afzalipour School of Medicine,
Kerman University of Medical Sciences, Kerman, Iran, Cardiovascular Research Center, Institute of Basic and Clinical Physiology Sciences,
2
Kerman University of Medical Sciences, Kerman, Iran, Endocrinology and Metabolism Research Center, Institute of Basic and Clinical
3
Physiology Sciences, Kerman University of Medical Sciences, Kerman, Iran, Gastroenterology and Hepatology Research Center, Institute of
4
Basic and Clinical Physiology Sciences, Kerman University of Medical Sciences, Kerman, Iran, Department of Physical Education, Faculty of
5
Medicine and Physiology Research Center, Kerman University of Medical Sciences, Kerman, Iran
ARTICLE INFO ABSTRACT
Article history: Background and Aim: Mitochondria play an important role in signaling and metabolic
Submitted: July 22, 2024 pathways in skeletal muscle. In this study, the effects of MitoQ supplementation alone and
Accepted: December 9, 2024 in combination with endurance training (ET) or high-intensity interval training (HIIT) were
Published Online: December 24, 2024 investigated in relation to the process of mitochondrial quality control in the gastrocnemius
muscle of male rats.
Keywords: Methods: Animals were assigned into 6 groups (n = 7): Control, MitoQ, ET, ET + MitoQ, HIIT,
MitoQ and HIIT + MitoQ. The gene and protein expression were quantified using real-time polymerase
Exercise training chain reaction (2 -ΔΔCT ) and Western blot analysis, respectively. Statistical analysis was performed
Fission using one-way analysis of variance.
Fusion Results: ET significantly increased protein expression of dynamin-related protein 1 (DRP1) and
Mitochondrial dynamic mitofusin1 (MFN1) and gene expression of optic atrophy Type 1 (Opa1) in skeletal muscle, when
compared to the control group (p < 0.001). HIIT only increased MFN1 protein expression compared
*Corresponding author: to the control group (p < 0.0001). MitoQ in combination with HIIT significantly increased protein
Beydolah Shahouzehi expression of DRP1 and MFN1 compared to MitoQ alone (p < 0.01).
Gastroenterology and Hepatology Research Conclusion: In sum, exercise training can affect mitochondrial dynamics by changing the factors
Center, Institute of Basic and Clinical involved in the fission and fusion process, and ET can improve training capacity in skeletal muscle
Physiology Sciences, Kerman University of by modulating expression of OPA1 and MFN1. While MitoQ supplementation alone did not
Medical Sciences, Kerman, Iran. significantly alter the mitochondrial fission-fusion process, its combination with HIIT appeared
Email: bshahouzehi@gmail.com to elevate the expression of DRP1, suggesting a potential synergistic effect that warrants further
investigation. Future studies should delve into the mechanisms by which MitoQ and exercise-
© 2024 Author(s). This is an Open-Access induced stress affect mitochondrial quality control, particularly in the context of redox modulation
article distributed under the terms of the and signaling pathways that govern mitochondrial plasticity.
Creative Commons Attribution-Noncommercial
License, permitting all non-commercial use, Relevance for Patients: Combining MitoQ with exercise training may enhance mitochondrial
distribution, and reproduction in any medium, function, potentially improving muscle health in patients.
provided the original work is properly cited.
1. Introduction
Regarded as dynamic organelles, mitochondria change their shape and structure in
response to various metabolic stimuli. Mitochondrial structure is mainly regulated by
fusion and fission cycles. These processes are tightly regulated to ensure a balance.
Dysregulated fusion and fission of mitochondria is an important mechanism in the
development of some diseases [1]. Mitochondrial dysfunction results in chronic
DOI: http://doi.org/10.36922/jctr.24.00044

